Hypothesis: Humans are adapted to starvation and the metabolically related low CHO diet/INSINH state. On the other hand, cancers in large cohorts of people in developed countries are under high chronic insulin stimulation and are largely unexposed to the unfamiliar INSINH state. These cancers will sensibly express a wide range of molecular and metabolic vulnerabilities to INSINH. However, they may express an equally wide range of adaptive mutations. Investigators have confirmed both vulnerability of some cancer types to inhibition by added ketone bodies as well as continued uninhibited cancer growth with added KBs in other cancers.
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